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Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might eventually be mediated by modulation of MAPK signaling cascades.
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Journal of Natural Medicines [03 Sep65 1: Find all citations in this journal default. Or filter your current search. Find all citations in this journal default. Morin treatment also leo the number of trabecular bones in DEX-induced rats. Journal of Natural Medicines [24 Jul72 4: Gene Ontology GO Terms. Abstract Ginkgo biloba, an herbal medication, is capable of lowering glucose, fat, and lipid peroxide in diabetic patients.
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Here, we uncovered the therapeutic effect of morin against osteoporosis and demonstrated its suppressive action on the MAPK pathway in this disease.
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Body mineral density BMD kei determined at proximal femurs using dual energy X-ray absorptiometry. Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription. Pathological examination was performed by hematoxylin and eosin staining. Our data suggest that GBE has the potential to prevent insulin resistance and is a promising anti-diabetic drug.
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CitePeer Related Articles http: It should be noted that, unlike rosiglitazone, GBE did not stimulate excessive glucose uptake as it improved glucose tolerance. The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively.
In the current study, we tested the hypothesis that Ginkgo biloba extract GBE prevented hyperinsulinism-induced glucose intolerance in hepatocytes. Body weight was 111418 monitored. To better show its efficacy, we included a control group that was treated with rosiglitazone, a type of thiazolidinedione TZD.
CitePeer Related Articles http: It is said that GBE treatment could avoid drug-induced obesity.